TableĀ 3

Clinical, angiographic, outcome and follow-up data of embolized cases

Clinical featuresImagingTypeJugular vein statusOutcomeFollow-up*
Neonates
1FDPAHChoroidalPatentImproved7
2CHFCDMuralPatentImproved8
3CHFCDChoroidalPatentDied1 day
4CHFH, WMMuralPatentDied8 days
5FDMBChoroidalAtresiaDied2 days
Infants
6CHFHMuralPatentImproved120
7MC, DDHMuralPatentImproved24
8MCH, ATMuralStenosisImproved36
9MC, DD, SzH, AT, SCMuralStenosisDeteriorated84
10MC, SzHMuralStenosisImproved12
11DD, SzH, WMMuralAtresiaDeteriorated18
12DD, SzH, BCChoroidalAtresiaDeteriorated7
13MCHChoroidalPatentStable6
14MC, DDH, ATMuralPatentImproved6
15MC, DD, SzH, SC, WMChoroidalAtresiaDeteriorated6
16MC, DD, SzH, AT, WMChoroidalAtresiaDeteriorated12
Children
17HA, VH, IVHChoroidalPatentImproved6
18Sz, DDH, ATMuralPatentImproved9
19HA, V, VLHChoroidalStenosisImproved18
20E, P, FVHMuralStenosisImproved6
21Sz, P, FVHMuralPatentImproved6
Adults
22HA, FVH, BCMuralStenosisImproved192
23HAHChoroidalPatentDied2 days
  • *Follow-up is given in months unless specified otherwise.

  • AT, cerebral atrophy; BC, basal ganglia calcification; CD, cardiomegaly; CHF, congestive heart failure; DD, developmental delay; E, epistaxis; FD, feeding difficulty; FV, prominent facial veins; H, hydrocephalus; HA, headache; MB, melting brain; MC, macrocrania; P, proptosis; PAH, pulmonary arterial hypertension; SC, subcortical calcification; Sz, seizure; V, vomiting; VL, visual loss; WM, cerebral white matter signal change.